Researchers at the University of Pennsylvania found that the protein TRF2 is essential for muscle stem cells to maintain their identity. Without it, the cells remain alive but lose the genetic program needed for repair. Injuries then heal with fat and scar tissue instead of functional muscle.
This offers a new target for muscular dystrophy treatments by preserving regenerative capacity in struggling muscles. The study used mouse models, so human application remains theoretical, and it is not yet a clinical therapy.
more: https://www.sciencedaily.com/releases/2026/11/261111221643.htm
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